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Home > Anti-aging Research > Zyflamend
Zyflamend
Specific Recommendation:
News and Research:
- 8
natural pain relievers - MSNBC, 9/14/08 -
"Capsaicin: For arthritis, shingles, or neuropathy ... InflaThera or
Zyflamend: For arthritis ... Aquamin: For osteoarthritis ... SAM-e (S
adenosylmethionine): For osteoarthritis ... Fish oil: For joint pain from
arthritis or autoimmune disorders ... Methylsulfonyl-methane (MSM): For
osteoarthritis"
- Herbal
extract may treat prostate cancer - MSNBC, 11/27/05 -
"Zyflamend has the ability, in culture
at least, to reduce prostate cancer cell growth by as much as 78 percent and
induce cancer cell death or “apoptosis,”"
-
Herbal COX Inhibitor Acts Against Prostate Cancer Cells - Medscape,
11/25/05
-
Columbia Study Suggests Benefits Of Herbal Extract In Early Treatment Of
Prostate Cancer - Science Daily, 11/13/05 -
"Zyflamend®, a unique herbal extract
preparation, suppresses the growth of prostate cancer cells and induces
prostate cancer cells to self-destruct via a process called “apoptosis"
- Herb Mix Nixes Prostate Cancer in Lab - WebMD, 12/13/02 -
"A number of recent studies point to COX-2 as an important factor in cancer-cell growth. In the Columbia studies, Zyflamend decreased
COX-2 activity about as well as a potent COX-2-inhibiting drug ... The herbal mix is called Zyflamend, from New Chapter Inc., and has 10 herbs: holy basil, turmeric, ginger, green tea, rosemary, hu zhang, Chinese goldthread,
barberry, oregano, and Scutellaria biacalensis"
Abstracts:
-
Zyflamend
Reduces the Expression of Androgen Receptor in a Model of Castrate-Resistant
Prostate Cancer - Nutr Cancer. 2011 Sep 29 - "These results demonstrated
that Zyflamend inhibited IGF-1-stimulated cell growth, IGF-1R expression, and
androgen receptor expression and its nuclear localization, but these effects
were not dependent upon phosphatidylinositol 3-kinase/pAKT signaling. In
conclusion, Zyflamend decreased cell proliferation and inhibited IGF-1R and
androgen receptor expression in a phosphatidylinositol 3-kinase/pAKT independent
manner"
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Zyflamend
suppresses growth and sensitizes human pancreatic tumors to gemcitabine in an
orthotopic mouse model through modulation of multiple targets - Int J
Cancer. 2011 Sep 20. doi: 10.1002/ijc.26442 - "Zyflamend inhibited the
proliferation of pancreatic cancer cell lines regardless of p53 status and also
enhanced gemcitabine-induced apoptosis. This finding correlated with inhibition
of NF-kB activation by Zyflamend and suppression of cyclin D1, c-myc, COX-2,
Bcl-2, IAP, survivin, VEGF, ICAM-1, and CXCR4. In nude mice, oral administration
of Zyflamend alone significantly inhibited the growth of orthotopically
transplanted human pancreatic tumors, and when combined with gemcitabine,
further enhanced the antitumor effects. Immunohistochemical and Western blot
analyses of tumor tissue showed that the suppression of pancreatic cancer growth
correlated with inhibition of proliferation index marker (Ki-67), COX-2, MMP-9,
NF-kB, and VEGF. Overall, these results suggest that the concentrated multiherb
product Zyflamend alone can inhibit the growth of human pancreatic tumors and,
in addition, can sensitize pancreatic cancers to gemcitabine through the
suppression of multiple targets linked to tumorigenesis"
-
Zyflamend
Mediates Therapeutic Induction of Autophagy to Apoptosis in Melanoma Cells -
Nutr Cancer. 2011 Jul 11 - "Zyflamend, a unique multiherbal extract preparation,
is a promising antiinflammatory agent that has also been suggested to regulate
multiple pathways in cancer progression. As Zyflamend contains ingredients that
can suppress tumor cell proliferation, invasion, angiogenesis, and metastasis
through regulation of inflammatory pathway products, we hypothesized that this
preparation might inhibit melanoma proliferation. To test this hypothesis, we
studied the effect of Zyflamend on melanoma proliferation. Here, we present that
Zyflamend inhibits melanoma growth by regulating the autophagy-apoptosis switch.
Based on the responsible molecular mechanisms of Zyflamend, our study highlights
the importance of the use of herbal preparations for the prevention and
treatment of cancer"
-
Zyflamend, a polyherbal preparation, inhibits invasion, suppresses
osteoclastogenesis, and potentiates apoptosis through
down-regulation of NF-kappa B activation and NF-kappa B-regulated
gene products - Nutr Cancer. 2007;57(1):78-87 -
"Zyflamend inhibited receptor activator of
NF-kappa B ligand-induced osteoclastogenesis, suppressed tumor
necrosis factor (TNF)-induced invasion, and potentiated the
cytotoxicity induced by TNF and chemotherapeutic agents, all of
which are known to require NF-kappa B activation. Zyflamend
suppressed NF-kappa B activation induced by both TNF and cigarette
smoke condensate. The expression of NF-kappa B-regulated gene
products involved in antiapoptosis (inhibitor-of-apoptosis protein
1/2, Bcl-2, Bcl-xL, FADD-like interleukin-1betaconverting
enzyme/caspase-8 inhibitory protein, TNF receptor-associated
factor-1, and survivin) and angiogenesis (vascular endothelial
growth factor, cyclooxygenase-2, intercellular adhesion molecule,
and matrix metalloproteinase-9) was also down-regulated by
Zyflamend. This correlated with potentiation of cell death induced
by TNF and chemotherapeutic agents. Overall, our results indicate
that Zyflamend suppresses osteoclastogenesis, inhibits invasion, and
potentiates cytotoxicity through down-regulation of NF-kappa B
activation and NF-kappa B-regulated gene products"
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